atra
- 网络全反式维甲酸(all-trans retinoic acid);全反式视黄酸;自动变速器再造协会(Automatic Transmission Rebuilders Association)
atra
atra
全反式维甲酸(all-trans retinoic acid)
...观察 龚维舜鲁厚清【摘要】:正国内自1986年开展全反式维甲酸(ATRA)治疗白血病以来,临床资料已经表明,ATRA治疗急性早 …
全反式视黄酸
经全反式视黄酸(ATRA)处理4d的细胞,调整浓度至1×106个/ml,接种到24孔培养板中(1ml/孔),加入1ml含有1.25mg/mlNB…
自动变速器再造协会(Automatic Transmission Rebuilders Association)
Automatic Transmission Rebuilders Association(ATRA) Battery Council International(BCI) Bear Automotive Bendix Bri...
全反维甲酸
全反维甲酸(ATRA)是体内具有重要生理活性的Vit A代谢产物之一,国内外一些报道表明:RA类化合物具有抑制细胞增殖、诱导 …
1
Recently, all-trans retinoic acid (ATRA) has been used as an antifibrosis drug in many experimental models.
近来较多的研究表明全反式维甲酸(ATRA)具有抗纤维化作用。
2
ATRA could inhibit HL60 cell proliferation, and with the increase in drug concentration, the effect of inhibiting the more significant.
ATRA明显抑制HL60细胞增殖,并随药物浓度的增加,抑制效果也越显著;
3
Thus, we studied whether maintenance therapy in ALL, like ATRA in APL , may be inducing terminal differentiation of ALL progenitors.
像在APL上使用ATRA一样,我们研究的所有维持疗法都可以诱导原生组织分化。
4
Results There were obvious differences in gene expression between human lung cancer cells before and after ATRA treatment.
结果在全反式维甲酸诱导前后的细胞之间存在明显的基因表达差异。
5
Cellular chemical staining and transmission electron microscope results also show that ATRA could induce HL60 cells to granulocyte cells.
细胞化学染色和透射电镜结果亦表明诱导后的HL60细胞向粒系分化。
6
METHODS CTX-dwas fractionated and purified from Naja naja atra venom by column chromatography and RP-HPLC.
方法应用柱层析及RP-HPLC,从眼镜蛇毒粗毒中分离纯化细胞素素(CTX)。
7
After ATRA and chemotherapy treatment, transcript levels gradually decreased to 1030 copies.
经过ATRA+化疗治疗后转录本水平又逐渐下降至1030。
8
ConclusionsThe model of ATRA-induced HL60 cells to granulocyte differentiation was successfully established.
结论建立了ATRA诱导HL60细胞向粒系分化的完整模型。
9
Objective: To study the anti-proliferation effects of ATRA on MB39 cells and the approach of its action.
目的:本文主要探讨ATRA对髓母细胞瘤细胞系MB39细胞增殖生存的影响及其作用途径。
10
OBJECTIVE To purify cytotoxin(CTX) from Naja naja atra venom, and to study its antitumor activity.
目的分离纯化眼镜蛇毒细胞毒素,测定其体内外抗癌作用。